Thursday, June 23, 2011

You can now experience a strong and healthy cardiovascular system

CoQuinone 30

have 
WHAT IS IT?
The energy that every cell needs to function is produced through a complex process in the mitochondria, an organelle within the cell often called the cellular powerhouse. Cells store energy in a molecule called adenosine-5-triphosphate or ATP. CoQuinone 30 contains advanced levels of Coenzyme Q10 (an essential part of the electron transport chain used to make ATP), which is synthesized and used by every cell in the body.


WHAT DOES IT DO?

Cells with the highest energy demands, such as in the heart, contain the highest levels of CoQ10. CoQ10 has been studied for years in the U.S., Europe, and Japan for its role in producing cellular energy and re-energizing the heart and other muscles.

- Includes advanced levels of Coenzyme Q10, which promotes a strong and healthy cardiovascular system and contributes to overall antioxidant protection.

- Supports energy metabolism in muscles


WHAT MAKES IT DIFFERENT?
- Clinical tests show that Coquinone 30 delivers CoQ10 in much higher quantities than from solid formulations or from competitive liquid formulations, making Coquinone 30 more bioavailable than other CoQ10 products.

- A potent antioxidant blend

- Patented formula: Contains both Coenzyme Q10 and alpha lipoic acid in a highly bioavailable formula

- Listed in the Physicians Desk Reference

- Pharmaceutical Quality: Effective and Science-based. C.A.P.P.S

- Laboratory tested, quality guaranteed, meets USP specifications for potency, uniformity, and disintegration


INGREDIENTS

Coenzyme Q10 
An energy coenzyme that plays an essential role in mitochondrial electron transport, and is thus fundamental for energy production in our cells.

Coenzyme Q10 is also an antioxidant. Its ability to quench free radicals helps maintain the structural integrity and stability of cell membranes (including intracellular membranes). It further serves to reduce oxidation of low-density lipoprotein (LDL) cholesterol.

Evidence suggests that the most important antioxidant activity of Coenzyme Q10 involves regeneration of Vitamin E. CoQ10 is also called ubiquinone, a name that signifies its ubiquitous (widespread) distribution in the human body. Highest levels of Coenzyme Q10 are found in the heart, liver, kidney, and pancreas.

CoQ10 supplementation has been shown to have therapeutic benefits for several diseases. Some of the best-documented effects involve cases of heart failure, ischemic heart disease, certain muscular dystrophies, hypertension, and periodontal disease.

CoQ10 is synthesized in all cells of the body (but particularly liver cells). It is also absorbed from the foods we eat. Major sources of dietary CoQ10 include meats, fish, and vegetable oils (particularly soybean, sesame, and rapeseed oils). Vegetables are generally low in CoQ10, with the exception of spinach and broccoli.

As aging occurs, the body's ability to synthesize CoQ10 diminishes significantly. Deficiencies may also result from reduced assimilation from dietary sources.

Coenzyme Q10 supplements are generally considered safe and are best absorbed by the body when taken with foods. The usual maintenance dose is 10-30 mg per day, although higher doses are used therapeutically for the treatment of heart and blood vessel disease.


Alpha lipoic acid (ALA), also known as thioctic acid
A vitamin-like antioxidant sometimes referred to as the "universal antioxidant" because it is both fat and water-soluble. Alpha Lipoic Acid is capable of regenerating several antioxidants back to their active states, including vitamin C, vitamin E, glutathione, and coenzyme Q10.

Several experimental and clinical studies have shown that alpha lipoic acid has potential therapeutic uses in diabetes, atherosclerosis, cataracts, heavy metal poisoning, neuro-degenerative diseases, and HIV infection.

Alpha lipoic acid is produced naturally by most organisms (including humans). It is also present in many foods. Potatoes provide a particularly significant dietary source.

Currently no RDI value has been set, nor has the issue been adequately studied. Some researchers believe that the amount needed for therapeutic antioxidant activity exceeds that produced by our bodies or consumed in a normal diet. As such, alpha lipoic acid is a strong candidate for dietary supplementation. Maintenance doses of 10-25 mg per day have been suggested and doses of up to several hundred milligrams per day have been used therapeutically.


Medium-Chain Triglycerides (MCTs)
Derived from coconut oil and are much more readily digested, absorbed, and metabolized than either animal fats or vegetable oils, which contain mainly long-chain triglycerides. MCTs are an excellent delivery system for fat-soluble elements, such as vitamin E, especially for individuals with fat malabsorption disorders.

(Note: MCTs do not contribute to a rise in blood cholesterol.)

Glycerin Monooleate
A polar lipid that functions as a humectant and solvent. It is prepared by the esterification of glycerin with fatty acids (chiefly oleic acid).

Glycerin
Functions as a humectant and solvent and is essential to the pliability and stability of gelcaps.

Annatto
Seed extract is a naturally occurring pigment that functions as both an opacifier and a food coloring agent.

Titanium Dioxide
A naturally occurring pigment that functions as an opacifier and food coloring agent. It occurs naturally in minerals, and is used in a wide variety of consumable products.


USANA CoQuinone 30 for cardiovascular health


PLEASE WATCH OUT FOR MORE POST, I WILL BE PROMOTING THE BEST SUPPLEMENTS WHICH YOU CAN TAKE TO PREVENT DISEASES AND ACHIEVE EXTRAORDINARY HEALTH.
HAVE A BEAUTIFUL LIFE AND EXPERIENCE TRUE HEALTH


References
1. Sarter B. J Cardiovasc Nurs 2002;16(4):9-20.
2. Langsjoen PH, Langsjoen AM. Biofactors 1999;9:273-84.
3. Soja AM, Mortensen SA. Ugeskr Laeger 1997;159:7302-08.
4. Satta A, et al. Clin Ther 1991;13:754-57.
5. Kamikawa T, et al. Am J Cardiol 1985;56:247-51.
6. Littaru GP, Battino M, Folkers K. Handbook of Antioxidants. New York: Marcel Dekker; 1996.
7. Kagan VE, Nohl H, Quinn PJ. Handbook of Antioxidants. New York: Marcel Dekker; 1996.
8. Alleva R, et al. Mol Aspects Med 1997;81(Supp):S105-12.
9. Kontush A, et al. Biochim Biophys Acta 1995;1258:177-87.
10. Nohl H, Gille L. Z Naturforsch [C] 1998;53:250-53.
11. Kagan VE, et al., J Lipid Res 1992; 33:385-97.
12. Bast A, Haenen GR. Biochim Biophys Acta 1988;963:558-61.
13. Kalen A, et al. Lipids 1989;24:579-84.
14. Aberg F, et al. Eur J Clin Invest 1998;28:235-42.
15. Mortensen SA, et al. Mol Aspects Med 1997;18(Supp):S137-44.
16. Palomaki A, et al. FEBS Lett 1997;410:254-58.
17. Cuomo J, Rabovsky A. Clinical Research Bulletin 2001. USANA Health Sciences.

No comments:

Post a Comment